Ptosis (eyelid)
Ptosis also known as Blepharoptosis is a drooping or falling of the upper eyelid. The drooping may be worse after being awake longer when the individual's muscles are tired. This condition is sometimes called "lazy eye", but that term normally refers to the condition amblyopia. If severe enough and left untreated, the drooping eyelid can cause other conditions, such as amblyopia or astigmatism. This is why it is especially important for this disorder to be treated in children at a young age, before it can interfere with vision development.
The term is from Greek πτῶσις – "a fall, falling".
Signs and symptoms
Signs and symptoms that are typically seen in this condition include in the following.- The person eyelid/s may appear to be drooping.
- Ptosis can also cause a person to appear tired from the droopy eyelids.
- Dry eyes is another symptoms due to the eyelid's inability to function effectively keeping the eyes from drying out.
- Obstruct vision may occur due to the sagging of the upper eyelids result which partially block the person's vision.
- A person tilts their head back to speak
- tiredness and aching around the eyes
- eyebrows are constantly being lifted to see properly
- eye tumor
- diabetes
- history of stroke
- cancer
- neurological disorders
- Older people.
Causes
Ptosis can be caused by the aponeurosis of the levator muscle, nerve abnormalities, trauma, inflammation or lesions of the lid or orbit. Dysfunctions of the levators may occur as a result of autoimmune antibodies attacking and eliminating the neurotransmitter.
Ptosis may be due to a myogenic, neurogenic, aponeurotic, mechanical or traumatic cause, and it usually occurs isolated, but may be associated with various other conditions, like immunological, degenerative, or hereditary disorders, tumors or infections.
Acquired ptosis is most commonly caused by aponeurotic ptosis. This can occur as a result of senescence, dehiscence or disinsertion of the levator aponeurosis. Moreover, chronic inflammation or intraocular surgery can lead to the same effect. Also, wearing contact lenses for long periods of time is thought to have a certain impact on the development of this condition.
Congenital neurogenic ptosis is believed to be caused by Horner's syndrome. In this case, a mild ptosis may be associated with ipsilateral ptosis, iris and areola hypopigmentation and anhidrosis due to paresis of the superior tarsal muscle. Acquired Horner syndrome may result after trauma, neoplastic insult, or even vascular disease.
Ptosis due to trauma can ensue after an eyelid laceration with transection of the upper eyelid elevators or disruption of the neural input.
Other causes of ptosis include eyelid neoplasms, neurofibromas or the cicatrization after inflammation or surgery. Mild ptosis may occur with aging.
A drooping eyelid can be one of the first signals of a third nerve palsy due to a cerebral aneurysm, that otherwise is asymptomatic and referred to as an oculomotor nerve palsy.
Drugs
Use of high doses of opioid drugs such as morphine, oxycodone, heroin, or hydrocodone can cause ptosis. Pregabalin, an anticonvulsant drug, has also been known to cause mild ptosis.Mechanism and Pathophysiology
Different trauma can cause and induce many of different mechanisms. For example, myogenic ptosis is when there's a direct injury to the levator muscle and/or Müller's muscle. On the other hand, neurogenic ptosis occur during closed head injuries, traumatically-introduced neurotoxin and botulinum toxin, which is caused by the effect those factors had in the CNNIII or the sympathetic pathway. Mechanical ptosis can also occur due to scarring tissue restricting the patient's eyelid excursion or it could have potentially created a mass that are weighing down the patient's lid. Another mechanism can be explained due to the disturbance of the oculomotor nerve causing the levator palpebrae to weaken results in the eyelid being droopy. Ptosis can also occur in a patient with brain tumors due to the pressure of the third nerve or also known as the sympathetic nerve on the brain stem, causing the eyelid to droop.Pathology
is a common neurogenic ptosis which could be also classified as neuromuscular ptosis because the site of pathology is at the neuromuscular junction. Studies have shown that up to 70% of myasthenia gravis patients present with ptosis, and 90% of these patients will eventually develop ptosis. In this case, ptosis can be unilateral or bilateral and its severity tends to be oscillating during the day, because of factors such as fatigue or drug effect. This particular type of ptosis is distinguished from the others with the help of a Tensilon test and blood tests. Also, specific to myasthenia gravis is the fact that coldness inhibits the activity of cholinesterase, which makes possible differentiating this type of ptosis by applying ice onto the eyelids. Patients with myasthenic ptosis are very likely to still experience a variation of the drooping of the eyelid at different hours of the day.The ptosis caused by the oculomotor palsy can be unilateral or bilateral, as the subnucleus to the levator muscle is a shared, midline structure in the brainstem. In cases in which the palsy is caused by the compression of the nerve by a tumor or aneurysm, it is highly likely to result in an abnormal ipsilateral papillary response and a larger pupil. Surgical third nerve palsy is characterized by a sudden onset of unilateral ptosis and an enlarged or sluggish pupil to the light. In this case, imaging tests such as CTs or MRIs should be considered. Medical third nerve palsy, contrary to surgical third nerve palsy, usually does not affect the pupil and it tends to slowly improve in several weeks. Surgery to correct ptosis due to medical third nerve palsy is normally considered only if the improvement of ptosis and ocular motility are unsatisfactory after half a year. Patients with third nerve palsy tend to have diminished or absent function of the levator.
When caused by Horner's syndrome, ptosis is usually accompanied by miosis and anhidrosis. In this case, the ptosis is due to the result of interruption innervations to the sympathetic, autonomic Muller's muscle rather than the somatic levator palpebrae superioris muscle. The lid position and pupil size are typically affected by this condition and the ptosis is generally mild, no more than 2 mm. The pupil might be smaller on the affected side. While 4% cocaine instilled to the eyes can confirm the diagnosis of Horner's syndrome, Hydroxyamphetamine eye drops can differentiate the location of the lesion.
Chronic progressive external ophthalmoplegia is a systemic condition that occurs and which usually affects only the lid position and the external eye movement, without involving the movement of the pupil. This condition accounts for nearly 45% of myogenic ptosis cases. Most patients develop ptosis due to this disease in their adulthood. Characteristic to ptosis caused by this condition is the fact that the protective up rolling of the eyeball when the eyelids are closed is very poor.
Diagnosis
The doctor will first perform a physical exam with questions asking about your medical history. This is to distinguish if the condition is hereditary or not. The doctor will then start with a slit lamp exam after you've explained how often your eyelids droop and how long each episode occurred. The slit lamp exam is done with the doctor uses a high-intensity light allowing them to take a closer look at the patient's eyes. The doctor can also perform a test known as the Tensilon test. The Tensilon test is when the doctor injects the drug Tensilon into your vein. The doctor then monitors your eyelids to see signs of improvements with the drug.Another comprehensive eye exam can be performed for the proper diagnosis is a visual field test. A visual field test may be performed to evaluates the superior vision assessing the quality of the patient's vision and how much is the ptosis affecting it. Since nerve damage is one of the causes for ptosis the ophthalmologist will check the patient's pupil for abnormalities. Along with checking the patient's pupil, the doctor will also check the patient's muscle function by having the patient look around.
The ophthalmologist may also measure the degree of the eyelid droop by measuring the marginal reflex distance. The marginal reflex distance is the distance between the center of the pupil and the edge of the upper lid. Along with the marginal reflex distance, the ophthalmologist may also examine the strength/ function of the patient's levator muscle. The ophthalmologist will do this test by holding the frontalis muscle. After holding the forehead the ophthalmologist will then measure how far the eyelid travels when the patient is gazing down.
Through these tests, the ophthalmologist will then diagnose if the patient has ptosis and what type of ptosis. After diagnosing the types of ptosis the ophthalmologist will then decides if the patient is a good candidate for surgery.
Classification
Depending upon the cause it can be classified into:- Neurogenic ptosis which includes oculomotor nerve palsy, Horner's syndrome, Marcus Gunn jaw winking syndrome, third cranial nerve misdirection.
- Myogenic ptosis which includes oculopharyngeal muscular dystrophy, myasthenia gravis, myotonic dystrophy, ocular myopathy, simple congenital ptosis, blepharophimosis syndrome
- Aponeurotic ptosis which may be involutional or post-operative
- Mechanical ptosis which occurs due to edema or tumors of the upper lid
- Neurotoxic ptosis which is a classic symptom of envenomation by elapid snakes such as cobras, kraits, mambas and taipans. Bilateral ptosis is usually accompanied by diplopia, dysphagia and/or progressive muscular paralysis. Regardless, neurotoxic ptosis is a precursor to respiratory failure and eventual suffocation caused by complete paralysis of the thoracic diaphragm. It is therefore a medical emergency and immediate treatment is required. Similarly, ptosis may occur in victims of Botulism and this is also regarded as a life-threatening symptom
- Pseudo ptosis due to:
- Lack of lid support: empty socket or atrophic globe.
- Higher lid position on the other side: as in lid retraction
Treatment
Aponeurotic and congenital ptosis may require surgical correction if severe enough to interfere with the vision or if appearance is a concern.Treatment depends on the type of ptosis, and is usually performed by an ophthalmic plastic surgeon or a reconstructive surgeon who specializes in diseases and problems of the eyelid.
If the condition occurs in a child, then the doctor will delay the surgery until the patient is 4 or 5 years old. If the patient is under the recommended age for surgery, then the doctor will test if occlusion therapy can compensate for the patient's impeded vision. The reason for delaying the surgery until the patient is at least 4-5 years of age is due to the delay for the frontonasal and upper face to complete their complex growth. After this complex growth is complete, the doctors will be able to obtain a more accurate measurement of the conditions. However, if the patient's vision impediment worsens or proves unresponsive to the occlusion therapy, then surgery will be needed sooner.
Surgical procedures include:
- Levator resection
- Müller muscle resection
- Frontalis sling operation
- Whitnall Sling
Frontalis sling surgery is considered the most effective surgical treatment for moderate to severe congenital ptosis. Many different materials can be used for the surgery, though it is currently unclear which material has the highest success rate.
The Levator Resection and Advancement surgery should only be considered for patients who are experiencing a levator function less than or equal to 5 mm. The levator function is a measurement of the distance that the eyelid travels starting with the downgaze moving to the upgaze without moving the frontalis muscle.Although this procedure can be completed through two different approaches, the internal and the external, the external approach allows the surgeons to obtain a better view of the surgical site during the procedure. The surgeon will begin with an incision on the eyelid. Once the levator has been exposed, the surgeon either fold it or cut it off before suturing it to the tarsal plate. During this procedure, it is up to the surgeon to decide the height and the contour of the patient’s eyelid, with input from the patient.
The Whitnall Sling procedure is done with an incision from the levator to the Whitnall ligament. Then the surgeon will suture the Whitnall’s ligament connecting it to the superior tarsal edge. This procedure most likely is done if the patients are concerned about cosmetic appearance. The Whitnall Sling procedure is able to provide a better cosmetic result because the procedure is able to keep the Whitnall’s ligament intact. This allows the support of the lacrimal gland and temporal eyelid to be maintained.
Despite the gains that the patient can obtain from the surgeries, there are risk factors. After the surgery, the patient may experience asymmetrical eyelids. If the surgery was not done carefully, the patient may experience dry eyes due to the eye no longer fully closing. The patient may also experience bleeding after the surgery and infections, if the surgical site is not taken care of properly.On rare occasions, the patient will experience a loss in eyelid movement.
Non-surgical modalities like the use of "crutch" glasses or ptosis crutches or special scleral contact lenses to support the eyelid may also be used.
Ptosis that is caused by a disease may improve if the disease is treated successfully, although some related diseases, such as oculopharyngeal muscular dystrophy, currently have no cures.